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Different types of anabolic steroids bind to the androgen receptor with different affinities, depending on their chemical structure. The most effective of these are steroids that cross the blood-brain barrier readily—for example, estrogens. Most other steroids (and their derivatives) are more difficult to cross that way. A small number of the most commonly used steroids (or their derivatives) are known to be nonmetabolically active at the blood-brain barrier, as described below. For these reasons, it is important to distinguish between these types of steroids: Steroids that cross the blood-brain barrier are non-androgenic. A lot of research is under way to better understand non-androgenic anabolic steroid binding in the brain. Steroids that are metabolically active at the brain endocrine gland do not cross the blood-brain barrier. Examples would be estradiol and progesterone, which have a high affinity for the androgen receptor and are not metabolized by the liver. Some steroids and their derivatives are not metabolized and do not cross the blood-brain barrier. For example, the use of non-androgenic anabolic steroids as a means to exercise anorectic women may result in adverse metabolic effects (especially weight gain and development of endocrine abnormalities such as male pattern hair loss) in men, although the long term effect of this practice is unclear. Steroids with low affinity for the androgen receptor have low binding affinities and are not very effective at the blood-brain barrier. Steroid pharmacology What is happening in a human is anabolic steroid action: increases in muscle mass and strength, decreases in fat mass and fat distribution, increases in muscle metabolism, and decreases in appetite and food intake. The majority of the anabolic steroids found in the human body are, in order from most to least potent, aromatized. The anabolic steroids most commonly associated with increased muscle mass are testosterone and human chorionic gonadotropin (hCG). Other anabolic steroids most commonly observed in decreased muscle mass are androstenedione, dehydroepiandrosterone sulfate, and testosterone. In some cases, there may be more or less potent anabolic steroids on either side of the blood-brain barrier that bind specifically to the androgen receptor. In general, compounds classified as non-anabolic steroids bind to the androgen receptor equally. Steroids, their metabolites, and their androgen agonists are divided into three classes according to their mechanism. androgen Similar articles:

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